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Tuesday 24 May 2011

Atkins, glucose, weight and insulin metabolism.

Insulin dependent diabetes is a relative term; that is your sensitivity to insulin is much higher than the lady next to you labeled as non insulin dependent diabetic mellitus.

You may be both diabetic but the effects are as different as chalk and cheese between insulin dependent and non insulin dependent diabetics. Insulin responds to blood glucose but the most devastating effects are from the cell glucose in the acute phase and it's low in the insulin dependent and normal in the non insulin dependent group.

Leptin is only as useful as helping you to stop habituation. You have to indulge every now and then to so as to make sure your metabolic rate does not match your intake in case you believe in minimal intake diet, which we don’t. This will halt your BMR and stop your weight loss progress.

 I will therefore mention it only this once at a practical level. Remember our premise is the world is a Garden of Eden and has every micro nutrient to heal everything.
If you don’t start munching now you might not reach the right nutrient level by the time you need it to correct an inevitable genetic error somewhere in your system. Worse still, medicine might have not reached a level of competence of knowing how to deal with it, which is likely considering we know so little about normal genes let alone genes gone abnormal.

What is the fuss the body makes about insulin and glucose? To answer this question we have to start with the fact that the brain, an important organ for most of us, can only metabolize glucose. You have no glucose for a few minutes you get comatose.
The other side of keto acidosis is a little more difficult to explain in a blog. The primary problem is that an insulin dependent diabetic with glucose in the blood ten times normal has no glucose inside the cells to feed them. The glucose it outside and not available for metabolism. The rest of the cells can use ketones (fat) but the brain is starved.
This means that our most important organ is at risk; therefore the intricate mechanism to release fat from all stores and reserve glucose for the brain is vital. If on the other hand you have only insulin resistance, your insulin level in the all cells is adequate to meet the needs of even the brain (enough glucose inside the cell).
It is this combination of adequate cellular glucose and high insulin that packs your waist line with fat, therefore raising your circumference.

The high glucose has to be stored somewhere as it is detrimental to your health. In contrast it is the no cellular glucose, all fat and ketosis that Atkins advocates or promotes. I hope it makes a little more sense now why I will be prepared to come close to insulin dependent diabetic state but not ketosis. You starve the brain for one and you play havoc with fat metabolism. It works but it denies the brain the only energy source it can use.
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